Elderly Brain

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Elderly Brain



Na-eri!

N'ihi nnukwu ọchịchọ anyị nwere obere ebe dịnụ. EnhanceMindIQ na-ere ngwa ngwa. Ngwaahịa a dị ugbu a na ngwaahịa ugbu a ma dị ruo etiti abalị!

EnhanceMindIQ Brain Booster

Kedu ka anyị na-eziga gị

nkwutọ BOTTLE?

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Brain Boost


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Ihe ka ọtụtụ ná ndị mmadụ na-amalite ịhụ ọkwa nke "ike ụbụrụ" ma ọ bụ "arụmọrụ nke ọma" dịka nwata dị afọ iri atọ

Ọ bụrụ na ị na-arịa ụbụrụ na-adajụ gị

nwere ike ịhụ mgbaàmà ndị dị ka:

Nchekwa ncheta

Ichefu ihe dị ka igodo gị ma ọ bụ obere akpa

Ọ na-esiri ike itinye uche

Enweghị nchebara echiche

Enweghị mkpali

Ike dị ala

Enweghi ike ime uche

Brain Boost


AJỤ EKWU NDỊ A GA-EKWU ECHICHE GỊ

Ụbụrụ na-amalite ịkwụsị ncheta site na mmalite nke 30

Achịcha nkịtị anyị na-eri n'ụbọchị ọ bụla nwere ike ime ka ikike ụbụrụ anyị daa

Ọ bụrụ na ị gaghị agbaso nri ụbụrụ siri ike ma ọ bụ na-eme ihe na-eme kwa ụbọchị dịka EnhanceMindIQ, ụbụrụ gị ga-amalite ịda mbà na arụmọrụ, na-eme ka ị ghara ichefu ngwa ngwa ma lekwasị anya dị ka ị dị

Ịkụnye ụbụrụ gị na ihe niile e kere eke dị na EnhanceMindIQ bụ ụzọ kasị dịrị nchebe na nke kachasị mkpa iji mụbaa ike gị kwa ụbọchị

Ọganihu-Ụdị NWA

MGBE ECHICHE MERE

E gosipụtara ihe ndị dị na EnhanceMindIQ iji nyere aka mee ka ncheta ncheta dị na ndị ikom na ndị inyom nke dịka afọ niile. Ụmụ nwoke na ndị inyom na iri abụọ na iri abụọ tupu afọ iri atọ amalitere ịhụ ọganihu dị ukwuu na nchekwa ncheta ha na oge ụfọdụ na ncheta ncheta oge ogologo ebe ndị si 40 ruo 65 dị afọ 65 hụrụ oke mmụba na abụọ!

NWA OZI EBE EBEGHỊ

Ejiri usoro mmelite EnhanceMindIQ mee ka ị nwee ume ike ozugbo i were ya ma debe gị anya ma lekwasị anya n'ụbọchị. Oge nkwụsịtụ nke ehihie na-agwụsị ozugbo ị chere ahụmahụ Neuro! Ị ga-anọgide na-agbasi ike ma legide anya ruo mgbe ị mechiri anya gị maka ụra.

MGBE ECHICHE NA MGBE

Kwa ụbọchị, ahụ anyị na-agbanwe ma ụfọdụ n'ime ụbụrụ ụbụrụ anyị na-anwụ. Ka anyị na-etolite, anyị na-amalite mmetụta mmetụta nke nke a ma na-esiri ya ike ịnọgide na-elekwasị anya ma ọ bụ na-akpali akpali. Ị na-ahụ onwe gị na ị na-atụgharị igodo gị ma ọ bụ obere akpa ma ọ bụ na-echezọ ime obere ọrụ gburugburu ụlọ dị ka ị na-ekpofu ahịhịa ahụ ruo mgbe ụgbọala ahụ ga-abịa? Nke ahụ bụ n'ihi na ka anyị na-etolite ike uche anyị na-adabere na nrụgide ụbọchị ọ bụla anyị na-etinye na ụbụrụ anyị. Ọ bụ ya mere i ji chọọ EnhanceMindIQ - Ọ bụ otu n'ime usoro dị mma iji mee ka ụbụrụ gị nweta ihe oriri na vitamin niile ọ dị mkpa maka usoro ọgwụgwọ zuru oke ọ chọrọ ya ka ọ dị mma iji meziwanye akụkụ niile nke mmụba akpọrọ, gụnyere ilekwasị anya, ike, mkpụmkpụ na nchekwa ogologo oge, nsogbu mmezi nsogbu na ọtụtụ ihe ndị ọzọ.

EnhanceMindIQ - Nye Onwe Gị Ogige Echiche

EnhanceMindIQ bụ mgbakwunye ọhụrụ nke aghọwo ihe na-agaghị ekwe omume ịchọpụta n'ihi ihe kachasị mma eche echiche. A maara EnhanceMindIQ dị ka Nzuzo dị ike nke a na-ewere dịka "Ntinye ngosi". Nootropics site n'usoro na-eme ka ọrụ ntụrụndụ dị ka cognition, ncheta, ọgụgụ isi, mkpali, anya, na ịta ahụhụ site n'ịgbanwe ụbụrụ ntanetụ.

100% Nile Eke

Omume arụmọrụ kacha mma

100% Dị Mgbagwoju nke Phosphatidylserine

Ịrụ ọrụ ọma nke ụbụrụ ụbụrụ gị

Nnyocha Nnyocha nke Ụmụ Mmadụ

Kwadoro nchekwa na irè

Emepụtara na FDA GMP Facility

Nchekwa nchekwa

Ụlọ akwụkwọ nyocha ụlọ akwụkwọ

OTÚ EKWERE NA GỤỌ IHE

1 MGBE

Were 1 pill kwa ụtụtụ. Ọ bụ kpam kpam eke na 100% nchekwa

2 BRAIN-BOOST

Ngwakọta na-eme ka ihe ọ bụla dị na capsule ọ bụla na-arụ ọrụ ugbu a na-enye ụbụrụ gị ihe ndị dị mkpa iji mee ka elekwasị anya, ncheta na ike gị.

3 Nhọrọ

Bido ịhụ nsonaazụ dika:

Mee ka ebe nchekwa cheta

Mee ka ike

Mee ka ụbụrụ gị meghachi omume

Obi ụtọ na-enye obi ụtọ

Ịhụ ahụike zuru oke

Rịba ama nke ọma

Nchekwa ọma na ụbụrụ

Ọrịa na-etiti


Anyị nabata


Anyị na-elekọta


Okwu & Ọnọdụ | Amụma Nzuzo | Nlekọta ndị ahịa

Okwu FDA (US Food & Drug Administration) adịghị atụle ya. Ngwaghị ngwaahịa a iji chọpụta, gwọọ ma ọ bụ gbochie ọrịa ọbụla. Ihe ọmụma nke ụlọ ọrụ a ma ọ bụ ụlọ ọrụ a na-enye abụghị ihe mgbakwunye maka ndụmọdụ ihu na ihu gị na dọkịta gị, na ekwesịghịkwa ịkọwa dịka ndụmọdụ ndụmọdụ ahụike. Ihe nchoputa di na weebụsaịtị a bu ikpe ndi mmadu ma ghara ikwere na i ga enweta otu ihe.


Ekwenyeghị Nchịkwa na nchekwa nke EnhaneMindIQ. FDA naanị na-enyocha ihe oriri na ọgwụ ọjọọ, ọ bụghị ihe mgbakwunye dị ka ngwaahịa ndị a. Achọghị iji ngwaahịa ndị a chọpụta, gbochie, mesoo, ma ọ bụ gwọọ ọrịa ọ bụla. Pịa ebe a iji chọpụta ihe àmà nke ule, nyocha, nchọpụta, ma ọ bụ ọmụmụ na-akọwa abamuru, arụmọrụ ma ọ bụ nrụpụta nke EnhaneMindIQ dabeere na ọkachamara nke ọkachamara dị mkpa.

1. Bunn F, Goodman C, Sworm L, et al. Psychosocial factors that shape patient and carer experiences of dementia diagnosis and treatment: a systematic review of qualitative studies. PLOS Med. 2012;9(10):e1001331. www.ncbi.nlm.nih.gov/pmc/articles/PMC3484131/
Glutamate is another neurotransmitter that tends to decrease with age.[33][34][35] Studies have shown older subjects to have lower glutamate concentration in the motor cortex compared to younger subjects[35] A significant age-related decline especially in the parietal gray matter, basal ganglia, and to a lesser degree, the frontal white matter, has also been noted.[33][34] Although these levels were studied in the normal human brain, the parietal and basal ganglia regions are often affected in degenerative brain diseases associated with aging and it has therefore been suggested that brain glutamate may be useful as a marker of brain diseases that are affected by aging.[33]
Pharmacological treatment options for Alzheimer’s-related memory loss and other cognitive symptoms are limited, and none can stop or reverse the course of the disease. However, assessing cognitive impairment and identifying its cause, particularly at an early stage, offers several benefits.
Structured Unlearning: Marijuana May Impair Memory via the Brain's Non-Firing Cells
54. Rogalski E, Stebbins GT, Barnes CA, et al. Age-related changes in parahippocampal white matter integrity: a diffusion tensor imaging study. Neuropsychologia. 2012;50:1759–65. [PMC free article] [PubMed]
NIA Alzheimer’s and related Dementias Education and Referral (ADEAR) Center
76. Grundman M, Petersen R, Ferris S, for the Alzheimer's disease co‐operative study et al Mild cognitive impairment can be distinguished from Alzheimer's disease and normal ageing for clinical trials. Arch Neurol 20046159–66. [PubMed]
Decreasing levels of different serotonin receptors and the serotonin transporter, 5-HTT, have also been shown to occur with age. Studies conducted using PET methods on humans, in vivo, show that levels of the 5-HT2 receptor in the caudate nucleus, putamen, and frontal cerebral cortex, decline with age.[29] A decreased binding capacity of the 5-HT2 receptor in the frontal cortex was also found,[27] as well as a decreased binding capacity of the serotonin transporter, 5-HHT, in the thalamus and the midbrain.[31] Postmortem studies on humans have indicated decreased binding capacities of serotonin and a decrease in the number of S1 receptors in the frontal cortex and hippocampus as well as a decrease in affinity in the putamen.[32]
65. Marshall S, Demmings EM, Woolnough A, Salim D, Man-Son-Hing M. Determining fitness to drive in older persons: a survey of medical and surgical specialists. Canadian geriatrics journal: CGJ. 2012;15:101–19. [PMC free article] [PubMed]
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^ Jump up to: a b Harris SE, Deary IJ, MacIntyre A, et al. (October 2006). "The association between telomere length, physical health, cognitive ageing, and mortality in non-demented older people". Neurosci. Lett. 406 (3): 260–4. PMID 16919874. doi:10.1016/j.neulet.2006.07.055.
Prevalence of dementia and major subtypes in Europe: A collaborative study of population-based cohorts. Neurologic Diseases in the Elderly Research Group.
^ Jump up to: a b Barnes, C.; Burke, S. (2006). "Neural plasticity in the ageing brain". Nature Reviews Neuroscience. 7 (1): 30–40. PMID 16371948. doi:10.1038/nrn1809.
Before discussing normal age-related changes, it is necessary to mention a few common methodological challenges that plague the study of normal brain aging. As with all studies of aging, selection bias is a challenge- many potential study participants decline enrollment because they are either too healthy (and busy) or too ill.2 Additionally, people with limited social or financial support and functional limitations may be less likely to enroll in studies.3 This results in study findings that may not be generalizable to all older adults.
95. Heijer T, Vermeer S, Dijk E. et al Alcohol intake in relation to brain magnetic resonance imageing findings in older persons without dementia. Am J Clin Nutr 200480992–997. [PubMed]
Genes involved in long-term potentiation e.g. calmodulin 1 and CAM kinase II alpha.
The normal aging process is associated with declines in certain cognitive abilities, such as processing speed and certain memory, language, visuospatial, and executive function abilities.
16. Gallagher M, Rapp P. The use of animal models to study the effects of ageing on cognition. Annu Rev Psychol 199748339–370. [PubMed]
Cognitive change as a normal process of aging has been well documented in the scientific literature. Some cognitive abilities, such as vocabulary, are resilient to brain aging and may even improve with age. Other abilities, such as conceptual reasoning, memory, and processing speed, decline gradually over time. There is significant heterogeneity among older adults in the rate of decline in some abilities, such as measures of perceptual reasoning and processing speed.11 We will provide a current, brief overview of the neuropsychology of normal cognitive aging. Interested readers are directed to other sources for a more comprehensive review of this topic.4,12
Voxel-based morphometry of human brain with age and cerebrovascular risk factors.
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